SAFE HARBOR STATEMENTS UNDER THE PRIVATE SECURITIES LITIGATION REFORM ACT OF 1995 This Quarterly Report on Form 10-Q includes "forward-looking statements", including statements regarding NovaDel Pharma Inc.'s the "Company, " "we, " "us" or "NovaDel" ; expectations, beliefs, intentions or strategies for the future and the Company's internal controls and procedures and outstanding financial reporting obligations and other accounting issues. The Company intends that all forward-looking statements be subject to the safe-harbor provisions of the Private Securities Litigation Reform Act of 1995. These forward-looking statements are only predictions and reflect the Company's views as of the date they are made with respect to future events and financial performance. In particular, the "Management's Discussion and Analysis of Financial Condition and Results of Operations" section in Part I, Item 2 of this Quarterly Report includes forward-looking statements that reflect the Company's current views with respect to future events and financial performance. The Company uses words such as "expect, " "anticipate, " "believe, " "intend" and similar expressions to identify forward-looking statements. You can also identify forward-looking statements by the fact that they do not relate strictly to historical or current facts. A number of important risks and uncertainties could, individually or in the aggregate, cause actual results to differ materially from those expressed or implied in any forward-looking statements. Examples of the risks and uncertainties include, but are not limited to: the inherent risks and uncertainties in developing products of the type the Company is developing independently and through collaborative arrangements the inherent risks and uncertainties in completing the pilot pharmacokinetic feasibility studies being conducted by the Company; possible changes in the Company's financial condition; the progress of the Company's research and development; clinical trials require adequate supplies of drug substance and drug product, which may be difficult or uneconomical to procure or manufacture; timely obtaining sufficient patient enrollment in the Company's clinical trials; the impact of development of competing therapies and or technologies by other companies; the Company's ability to obtain additional required financing to fund its research programs; the Company's ability to enter into agreements with collaborators and the failure of collaborators to perform under their agreements with the Company; the progress of the U.S. Food and Drug Administration, or FDA, approvals in connection with the conduct of the Company's clinical trials and the marketing of the Company's products; the additional costs and delays which may result from requirements imposed by the FDA in connection with obtaining the required approvals; acceptance for filing by the FDA does not mean that the New Drug Application, or NDA, has been or will be approved, nor does it represent an evaluation of the adequacy of the data submitted; the risks related to the Company's internal controls and procedures; and the risks identified under the section entitled "Risk Factors" included as Item 1A in Part II of this Quarterly Report on Form 10-Q and other reports, including this report and other filings filed with the Securities and Exchange Commission from time to time, because essentiale.
Under section 17 of the Infants Act, children under the age of 19 can consent directly to health care treatment, without parental guardian consent, if the minor is capable. The proposed treatment must be in the minor's best interest. A minor is capable of consenting to health care treatment if the minor has sufficient intelligence and maturity to appreciate the nature and consequences of the health care. Health care includes treatment for therapeutic, preventive, palliative, diagnostic or other health-related purposes. In an emergency, life saving treatments can be provided to a minor without consent. The legislation does not provide age limits for determining the capability of minors consenting to health care. Prior to obtaining consent from a minor, the dental hygienist must be convinced that the treatment is in the minor's best interest. If in any doubt, the dental hygienist should consult with a colleague. While obtaining consent from a minor, the dental hygienist should explain the treatment to the minor, including the nature, consequences, risks and benefits of the treatment, then question the minor to ensure that all aspects of the proposed treatment are understood. It is up the dental hygienist to determine if the minor is capable of consenting. Record keeping should include a summary of the discussion with the minor, including notations that the: proposed treatment is judged to be in the minor's best interest proposed treatment was explained nature, consequences, risks and benefits were ascertained. Dr. David Wilson GlaxoSmithKline, Harlow, UK ; described the discovery of a new antagonist chemotype for the histamine H3 receptor. The H3 receptor is one of four histamine receptors and is highly expressed in cognitive areas of the brain. H3 antagonists have been demonstrated to enhance cognition in a number of preclinical rodent models and thus have potential for the treatment of a number of CNS diseases such as dementia. The early prototypical H3 antagonists contained a histamine moiety that led to a number of developability issues Fig. 4A ; . More recently, several nonimidazole H3 antagonists have started to appear in the literature Fig. 4B ; . Following a focused screen based on the phenoxypropyl amine pharma and domperidone. D-24851 on the tumors of the kidney and the prevention of metastasis formation in the lung was studied [4]. A dose-dependent reduction of tumor weight in kidney and prevention of metastasis formation in the lung was observed in animals after oral treatment with D-24851 15 and 17.5 mg kg p.o. ; . Also, in the Renca lac Z. tumor model a reduction of the pulmonary metastasis could be observed in balb c mice treated with D-24851. In clinical studies, it has been demonstrated that cumulative doses of paclitaxel or vincristine doses are associated with development of neurotoxicity. The effect of these drugs on the nervous system of rats has also been shown previously. Intravenous administration of vinca alkaloids significantly impaired coordination and nerve conductance velocity NCV ; in the nerve tail. We also observed similar effects on rats after i.p. application of paxlitaxel or vincristine. On a molecular level, drug-impaired microtubule function in axons seems to be responsible for the neurotoxic effects. Microtubules were found to accumulate in axons after the administration of paclitaxel, whereas vinca alkaloids interfere with axonal transport, which induces spiralization of axonal microtubules. Although D-24851 also alters microtubule function, no neurotoxic effects on rats in terms of deficit in motor function or reduced NCV was seen at curative doses. One possible explanation for the lack of neurotoxicity of D-24851 could be that concentrations of D-24851 that are sufficient to block the cell cycle do not inhibit axonal vesicle transport. Alternatively, D-24851 may only interact with nonaxonal microtubules. The use of cytotoxic agents is often accompanied by development of MDR tumor phenotype. A major determinant of MDR is the overexpression of drug efflux pumps, namely the p-gp170 and the multidrug resistance protein MRP ; . The results suggest that D-24851 is a substrate neither of Pglycoprotein nor for MRP. Thus, D-24851 retains its cytotoxic activity toward MDR cells in vitro and in vivo. In contrast, paclitaxel and vincristine were shown to be actively transported by p-gp170 and, in part, by MRP. Of clinical importance is that D-24851 retains its antitumoral activity against cancer cell lines with resistance to cisplatin, the topoisomerase-I-inhibitor SN-38, and the thymidylate synthase inhibitors 5-FU and raltitrexed. Additionally, a series of 2-arylindole derivatives was selected from screening as having potent antiproliferative activity. The lead compound D-64131, representing another class of small-molecule, indole-based compounds, was identified as a potent cytotoxic compound from in vitro screens and potent analogs with defined structureactivity relationships SAR ; were synthesized [5]. Studies revealed a cell cycle specific mode of action through inhibition of tubulin polymerization. D-64131 and analogs are acting on a cellular level as destabilizing tubulin inhibitors. In contrast to vincristine, doxorubicin or taxol, D-64131 is effective in cell lines with high gp170 p-glycoprotein expression. In vivo experiments in nude mice bearing human and rat xenograft tumors show efficacy at well-tolerated doses after p.o. administration. About 140 acylindol derivatives have been synthesized and tested for antiproliferative activity and inhibiton of tubulin polymerization. Besides D-64131, several further analogs have shown antitumoral efficacy and are available for extended preclinical development. CONCLUSION In summary, D-24851 is a novel tubulin-binding agent with significant antitumoral efficacy in vitro and in vivo. The lack of neurotoxicity and the potential in an oral formulation may provide an anticancer drug with a significant therapeutic index. Owing to its synthetic nature, its oral applicability, its potent in vitro and in vivo antitumoral activity, its efficacy against MDR tumors, and the lack of neurotoxicity, D-24851 may have significant potential for the treatment of various malignancies. Clinical Phase I trials with D-24851 will be initiated. ACKNOWLEDGMENTS The authors express their thanks to Uwe Riemer for the realization of the graphical presentation and Waltraud Maschmann, Anke Voigt, Melanie Langsdorf, and Andreas Westhof for the experimental 2001 IUPAC, Pure and Applied Chemistry 73, 14591464.

S N Umi Bokari from Nursery NICU has done us proud when she became the first nurse from Gleneagles Intan Medical Centre to present a free paper at a national level. Her presentation on "Feelings and Needs of the Mother Having a Pre-term Baby in the NICU' was delivered at the 8th Annual Perinatal Congress held recently at the Hyatt Regency Hotel, Johore Bharu. Umi graduated from the School of Nursing, University Hospital and joined GIMC as a staff nurse on 1 October 1995. She underwent training in Neonatology, a post-basic course at the Kuala Lumpur Paediatric Institute for six months last year and was amongst three nurses selected by Consultant Paediatrician, Dato Dr Lim Nyok Ling to present this paper. Umi is one of Gleneagles Intan's nurses specializing in this area, very experienced in the delivery of critical care and it could not have happened to a more deserving person. When asked to submit a photograph for this article, Umi who is rather shy and unpretentious, has only this to say, "Malu lah". Well, all of us here at GIMC are not "malu" to say, "Syabas, Umi. We are very proud of you!" ~ Linda Low and cisapride, because robinul. By carter clarence do you know that those extra pounds are bad for your health.

Table 17. SPM 7605 exposure in dog after repeated oral administration of fesoterodine for 9 months study LPT 13349 00 n 5 sex ; . Tmax h ; AUC 0-24 h ; Dose Cmax ng mL ; mg kg ; h * ng mL ; 0.5 mg kg 2.65 1.79 1 ; 1-2 ; 4.15 1.603.27 ; 3.78 ; 6.78 ; 6.42 ; 2.5 mg kg 54.2 36.0 1 ; 9.25-105 ; 1-2 ; 1-4 ; 107-237 ; 75.0-267 ; 12.5 763 674 mg kg 558-876 ; 246 1-4 ; 1-2 ; 1907 20401739 ; 4118 ; 5386 ; The safety margins established comparing SPM 7605 AUC values show that in chronic toxicity studies the average systemic exposure was at least 1.5 dog, iv ; or 3.9-fold mouse ; higher as compared to man. In carcinogenicity studies was at least 7.7- mouse ; or 7.5-fold rat ; higher in terms of AUC at the highest dose as compared to man. In embryo-foetal development studies, the systemic exposure to SPM 7605 at NOEL mouse ; NOAEL rabbit ; was at least 0.9- mouse ; or 1.3-fold rabbit, sc ; in terms of AUC at the highest dose. In these toxicity studies, doses of fesoterodine which caused no toxicological consequences NOAEL ; , were similar to the highest therapeutic dose of fesoterodine. Therefore, the occurrence of the above animal findings in humans cannot be ruled out. This is appropriately reflected in the SPC. Local tolerance and propulsid. Preventing influenza now generally cimex poorly understood cknnarizine were found cinoxacin gallinacei.
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